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I am particularly intersted in that since my brother also had (AML) leukemia over 5 years ago (before I knew about sodium chlorite), and I gave him pau 'd arco tea (a potent anti-fungal) and poke root tincture to take at the same time as his conventional treatment of which he only partly completed. You may ask, what does fungi have to do with leukemia? Let me direct you to a very interesting thoughts that discusses the implications of fungi and leukemia. View this piece by Dr. Holland in the following this post.

You will discover that many times a fungi infection can be mistaken for leukemia.

Here was another post, I did a few years ago on leukemia:


I just happened to be rereading a very interesting medical text from 1955, PATHOGENESIS OF CANCER by John Gregory MD. In one passage he is talking about leukemia. He writes:

"The various forms of leukemia which are found in man are imitated to a surprising degree in the mouse. It is becoming obvious that the leukemia of man and the lower animals are analogous. Drs. Fouth and Stubbs reported in 1931 that they had taken blood from leukemic chickens, injected it into other chickens and produced sarcoma at point of injection. Leukemia, a cancerous disease of the blood forming organs can be induced by a virus filtered from cancerous blood. This virus can localize not only in the blood forming organs, but under some conditions in other cells, and thereby produce new types of cancer."

I was up at my brother's house after reading the above passage and told him what I read
about leukemia. A light bulb went off in his head and he told me right before he came down with his leukemia, he was eating a ground turkey burger upon which a very thin sliver of a turkey bone had pierced his upper gum. After which, he developed all types of sores in his mouth and nostrals days later and, then soon after, experienced the initial affects of leukemia.

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I do darkfield blood work. I have finally came to the conclusion in the last few years that he has a form of fungi in his blood after examing his blood off and on. He must be carrying a heavy load as I have taken 3 blood samples and each sample seemed to contains this organism. Below this post you will see a photo micrograph of fungi, I saw growing in his blood from 1 hour on the slide to 24 hour growth.
After taking a third blood sample, I have unfailingly viewed in each of his blood samples this very unusual fungi growth. The first samples were viewed in blood that were not treated with my Acridine Orange/DMSO stain,nor was it observed under fluorescence, only darkfield, and this sample seemed to present the freest and fastest growing samples of the fungi when it was incubated. My subsequent next 2 blood samples were treated with a biologic stain, Acridine Orange/DMSO and though the fungi was still present in these latter two samples, its growth and viability seemed to be sharply checked, I presume because of the presence of Acridine orange which binds with DNA. I will attach three photos of what I am seeing in his blood in progressing time increments of initial sighting after 6 hours, and after 24 hours--all from the first unstained blood sample. The white rings of various shapes are red blood cells. The white, often granular blobs around the fungi are white blood cells.

It appears to me that this fungi belongs to the Alternaria family as viewed from this superb site:

starcentral.mbl.edu/microscope/p ... geid=24275

Note that I was very worried that my blood samples had been contaminated during the blood lanceting and slide preparation process, but with each succeeding blood specimen, I took, I was more and more careful with sterile technique. Nevertheless, the same fungi seems to be always in his blood.

The fungi in my brother's blood belongs to the Alternaria family. I found the below interesting case online that involves a man with that type of fungi infection:

"We report a case of cutaneous alternariosis in a 69-year-old male patient. Durin hospitalization for treatment of the skin disorder, acute myeloid leukaemia was diagnosed. He received multiple chemotherapeutic agents but the leukaemia remained refractory to therapy and the patient died. The clinical picture, diagnosis and treatment of cutaneous alternariosis will be discussed and a review of the literature regarding patients with haematological diseases will be given."

To translate in other words, a patient was admitted in a Greek hospital with a skin infection involving the alternaria fungus. While there, it was discovered he had acute myeloid leukaemia (as my brother had). My question is, which came first? The alternaria infection or his leukemia? I know the medical establishment probably thinks the patient's fungal infection was a result of the poor immune function resulting from the leukemia,,,,,,,,,,,but why not consider that the alternaria may have contributed to the leukemia? Mmmmmmmmm??

My theory in my brother's case is that he is carrying a perpetual fungal infection in his blood plasma and his immune system seems to be able to keep it well under control while he is in good health. Should his immune system become impaired,,,,,,,,,,he better look out! Funny thing. When I put his
blood on the slide, of course, his natural immunity ceases to be much of a factor which allows this fungus to start growing unchecked. If you notice in the photo micrographs that the last two photos actually has his white blood cells (the white granular material) surrounding the fungus in an attempt to do their job, even in the artificial environment of a microscope slide.

by David Holland, MD

I recently spoke with a nurse who was diagnosed, as an adult, with leukemia. She endured the chemotherapy regimen her doctors prescribed, only to suffer from a secondary fungal infection during that time. The intensity and duration of the antifungal treatment rivaled that of the chemotherapy. At any rate, she recovered from both afflictions and went back to work.

Later, as a result of another workup -- which included a liver biopsy -- for some returning symptoms she had, bad news was again brought up. "Your leukemia has returned," her oncologist told her, and he proceeded to lay out the next line of chemotherapy drugs she would have to take.

Given that her chances of dying were much higher now that her cancer had returned, she opted to get a second opinion on her biopsy before proceeding with her next round of chemotherapy. She took her tissue sample to another hospital, and what she was told there was absolutely stunning: "You don't have leukemia," remarked the pathologist, "what you have is a fungal infection!"

The scenario that her doctors figured was that her previous fungal infection had returned -- a total possibility. But for this nurse, more questions were raised. She thought, for example, "If they diagnosed my fungal infection as leukemia this time, is it possible that they were wrong the FIRST time? Was my leukemia really a fungal infection to begin with, and was my so-called 'secondary' fungal infection I had earlier really a full-blown manifestation of what originally might have looked like leukemia?"

Of course, she would never get answers to these questions, for to fully investigate thoughts like these might imply that a diagnostic error was made on the part of either her initial oncologist or pathologist.

Nevertheless, an intense six months later -- some of it spent in the hospital -- of high-dose, powerful antifungals finally achieved a cure for her fungal infection. Today, she is again back at work, exuding more than ever with compassion for her patients. It really struck me when she told me where she works, because in her case, her occupation may very well relate to what she had suffered over the past two years. It turns out that she works at a bone-marrow transplant center, and is in daily contact with children with leukemia.

Now, the thought of her "acquiring" something as grave as leukemia would almost be preposterous to some. But the temptation to scratch our heads and wonder about this is unbearable. What if she really did have a fungal infection -- and NOT leukemia -- her first time around? And if so, did she "catch" this from her precious little patients?

Fungal infections not only can be extremely contagious, but they also go hand in hand with leukemia -- every oncologist knows this. And these infections are devastating: once a child who has become a bone marrow transplant recipient gets a "secondary" fungal infection, his chances of living, despite all the antifungals in the world, are only 20%, at best.

And then the unthinkable thought arises: what if all of these children didn't even have leukemia, but rather a fungal infection, just as this nurse did? If doctors, in the 21st century, could mistake a fungal infection for leukemia in this nurse, could the same fate have fallen upon these children?

Doctors in general are not very good at diagnosing fungal infections because their medical school training is based so heavily on the role of bacteria and viruses in the area of infectious diseases. Fungi have been a forgotten foe ever since the advent of antibiotics. Once we had a drug that could kill bacteria, the interest in and the study of fungi fell to the wayside.

Laboratories display the same difficulty in diagnosing fungal infections: current tests for detecting the presence of fungi are both terribly scant and sorely antiquated.

Despite these training and technical inadequacies, there have been at least a few good reports that implicate the role of fungi in causing leukemia.

For example, in 1999 Meinolf Karthaus, MD, watched three different children with leukemia suddenly go into remission upon receiving a triple antifungal drug cocktail for their "secondary" fungal infections.(1)

Pre-dating that, Mark Bielski stated back in 1997 that leukemia, whether acute or chronic, is intimately associated with the yeast, Candida albicans. (2)

Finally, almost 50 years ago, Dr. J. Walter Wilson, in his textbook of clinical mycology, said that "it has been established that histoplasmosis and such reticuloendothelioses as leukemia, Hodgkin's disease, lymphosarcoma, and sarcoidosis are found to be coexistent much more frequently than is statistically justifiable on the basis of coincidence." (3)

Histoplasmosis is what we call an "endemic" fungal infection. It is most commonly acquired in regions surrounding the Ohio and Mississippi river valleys in the United States. One becomes ill by merely inhaling the tiny fungal spores of this fungus. (For more information on histoplasmosis and other endemic fungi, you can visit: www.doctorfungus.org/). Three similar reports like this over the span of 40 years should convince us to at least study the role of fungi in cancers like leukemia a little more thoroughly.

The late Milton White, MD., did exactly this. He fully believed that cancer is a "chronic, intracellular, infectious, biologically induced spore (fungus) transformation disease." (4) Using the proper isolation techniques (involving saline instead of formaldehyde as a tissue transportation medium between the operating room and the pathology lab), he was able to find fungal spores in every sample of cancer tissue he studied. His lifetime work has been routinely dismissed as nothing more than an unproven postulate.

Regardless, wouldn't you expect all of this information to make front-page headlines in every newspaper across the country, if not the world? Instead, every one of these findings was merely a brief mention -- only curious thoughts that one might entertain but never take seriously.

The fact is, if leukemia and fungal infections "co-exist" so frequently, and if an antifungal drug cocktail effectively cured at least these three children of their leukemia, then I say we put the brakes on right there. Is there a need to go any farther, except to more deeply investigate the need for antifungals in treating leukemia and not just the secondary infections that arise in the course of chemotherapy?

In his book, The Germ that Causes Cancer (iknowthecause.com), author and television host Doug Kaufmann asserts that not only fungi, but also foods play a role in the etiology of cancer. He has seen children become free of their documented leukemia once the child's parents simply changed the child's diet. Kaufmann's diet is base on the widely-published problem of mycotoxin contamination of our grain foods.

Grains such as corn, wheat, barley, sorghum, and other foods such as peanuts, are commonly contaminated with cancer-causing fungal poisons, or "mycotoxins." (5,6) One of them, called aflatoxin, just happens to be the most carcinogenic substance on earth. If this is indeed a problem, Kaufmann asserts, then cereal for breakfast and soda pop for dinner may not be conducive to a cancer-free lifestyle.

A case in point: in a grain-based diet, we consume, on average, from 0.15mg to 0.5mg of aflatoxin per day. (7) Further, he states, it is not the sugar alone that is the problem in our western diet, but the fungal toxins that are found in the sugary grains. More than once has Kaufmann interviewed a caller (on his health talk show) who absolutely craved peanut butter and popcorn just prior to their diagnosis of cancer.

Fungi are such a nuisance in carbohydrate foods in particular because fungi need carbohydrates to thrive. Therefore, it is rarer to see fungal contamination problems in foods like vegetables and high-protein foods.

Kaufmann goes on further to explain how even antibiotics may play a role in the disease process. Antibiotics destroy the normal, protective gut bacteria, allowing intestinal yeast and fungi to grow unchecked. These internal, gut yeast make toxins, too. This can lead to immune suppression, symptoms of any autoimmune disease, or even cancer. "If the onset of any symptom or disease- cancer included- was preceded by a course of antibiotics," he maintains, "then look for a fungus to be at the root of your problem."

Doug and I will be talking more about the role of fungi in cancer and other diseases, such as diabetes, in our upcoming seminars. Check out our website (iknowthecause.com) to find the location

Very very interesting Polo, thanks!

Here whats going on now.

I could get my dad to take the MMS for the two weeks we were out of the Hospital, mainly because he wants to drink orange juice and V8 all day. Doesn't mean that we wont be revisiting the idea. About a week after leaving the hospital he had another Bone Marrow Biopsy to see what the CPX Trial Chemo did. Well, if you remember originally his blood cells were
70% blast/cancerous. After the 3 rounds of chemo, and a little more than 3 weeks since his last does and while we were still in the hospital, his blast cells were down to 2%. About a week after being home they did another Bone Marrow Biospy and his blast cells were down to 0.1%. This is very good news. For the first time in more than a year his blood counts were normal all the way around, platelets, red blood, white blood, neutrophils. He hasn't had to have a transfusion of any sort since we left the hospital on the 25th or so of sept. Today is the 16th of October and we are back at the University of Washington's Cancer Center for 2 more rounds of the Trial Chemo. They say he is in remission now and should have another 6 month to a year to live. Thats alot better then 3 to 4 weeks. I did Hannas Tail Bone treatment 2 times since leaving the hospital. I would have done it more but after the last bone marrow biopsy he could even stand up on his own or anything. The doctor at Highline Medical Center should of taken him off the blood thinners for atleast a day before doing the biopsy, the results were internal bleeding and his whole leg swole up. He's getting better now though and can walk around with his walker a little better. Keep in mind that even though the Chemo might wipe out the AML, he still has MF, which I've also heard them say is MDS. He also has cancer on his liver.

Does anyone know of anyway to treat MDS or MF?

Thanks again for all of your input and keep all the ideas coming...we still have a long road ahead of us. Thanks again!

Thank you my friend, I hear you!

ditto what Ozone said ..

Mike,

the pills ...Acyclovir, Fluconcizole and Levofloxacin can be substituted by MMS. MMS can kill these germs. I got rid of my bacteria, viruses by MMS and the other pills did not work.

Amoxicillin/Clavulan 125MG (1) at 9am and (1) at 9pm

Acyclovir 800MG (1) at 9am and (1) at 9pm

Voriconazole 200MG (2) at 9am and (2) at 9pm

Lovenox Injections (1) at 9am and (1) at 9pm - Lovenox is a blood thinner for his blood clot.

Terazosin 1MG at 9am - to help with urination.


I have been on those meds except terazosin. The lovenox is a blood thinner, obviously they think he needs it at least for now.

the amoxicillin is for bacteria
acyclovir for viruses
voriconazole I think for parasites, not sure

MMS treats bacteria, viruses and parasites at the same time. So as soon as you are on MMS full dose he might not need them.
I tried my MMS first on my skin and then one drop per 8 oz a day, baby bottle protocol.

Hey, just wanted to give everyone an update on whats going on now. Its January 20th 2014 and everything is still going pretty good. We are back at the UW Cancer Center for the fourth round of Chemo, and so far the chemo has been keeping the AML in check. My dad is doing a lot better and is getting around on his own and all that good stuff. I just had him read through this whole discussion and I think it might of opened him up to the possibilities of MMS. He seems to be a lot more willing to try it after reading some of the things you guys have said here. After this round of chemo he has one more which will be in about 7 weeks I'd guess. After that I think there going to pretty much leave him alone for a while and see if the AML returns and at that point see what other options they can offer him. I'm hoping that we can get him on the MMS a month or so after the next/last round of chemo and erratic-ate the AML without needing any other options from the doctors. When that time comes I'll be back here for more updates and insight, thanks again to everyone partaking in this discussion.

One more thing...I'm over here at the UW Hospital and have been talking to all the Doctors about MMS. The main Doctor behind this CPX-351 trial has never heard of Chlorine Dioxide and is waiting for an email from me that explains it to him.

So, can any of you recommend a really good article explaining how MMS works and kills cancer? I'm looking for something bullet proof that when the doctor reads it he won't be able to deny. He's actually a pretty humble guy and really seems like he would like to know more about it. I can't say that about the rest of the doctors I talked too. I want to send him something that will make him have to study MMS, I mean if he's really in the business to help people it should peak his interest. I found plenty of articles on the net that I could send him but I wanted to see what you guys might have up your sleeves so we can make a joint effort to get this guys attention. Thanks!